Sugar Cure is a Herbal
blood sugar medicine /natural medicine for
diabetes that helps people recover from blood
sugar / diabetes problem forever. This herbal
Blood sugar cure medicine / supplement is 100% herbal preparation with 100% natural ingredients
and it
is very much effective and result oriented in curing and controlling blood sugar
/ diabetes problem forever.
Sugar Cure : Designed to restore
pancreatic function and to proliferate insulin
beta cells, Sugar Cure has been shown to gradually
and effectively lower blood-sugar levels and
increase insulin secretion naturally. In addition
Sugar Cure has been
shown to regulate carbohydrate metabolism which is the active fuel of
the body and is ordinarily the main source of energy of the tissue
cell, improve blood circulation, lower blood cholesterol and
increase immune response.
Sugar Cure is beneficial in :-
- Re-storing Pancreas for natural production of insulin by natural
process.
- Regulates Carbohydrates metabolism.
- Improves Blood Circulation.
- Adds Muscle mass.
- Adds Immune system response.
- Tones Nervous system.
- Gradually lowers Blood Sugar levels and re-stores it at normal
level.
About Blood Sugar:
Carbohydrate is the active fuel of the body and
is ordinarily the main source of energy of the
tissue cell. In the normal digestive process, food
sugars and starches (carbohydrates) are changed
into sugar glucose. This is stored in the form of
glycogen (animal starch) in the liver and muscles
for later use as a body fuel, at which time it is
reconverted into glucose. Blood sugar rises
somewhat after eating, and in healthy individuals
returns to normal levels in about an hour or two.
The amount of glucose in the blood is controlled
mainly by the hormones insulin and glucagon. Too
much or too little of these hormones (or if they
are somehow ineffective) can cause blood sugar
levels to fall too low (hypoglycaemia) or rise too
high (hyperglycaemia). Other hormones that
influence blood sugar levels are cortisol, growth
hormone and catecholamines (epinephrine and
norepinephrine).
The pancreas, a gland in the upper abdomen is
responsible for producing insulin and glucagon.
The pancreas is dotted with hormone-producing
tissue called the islets of Langerhans, which
contain alpha and beta cells. When blood sugar
rises after a meal, the beta cells release
insulin. The insulin helps glucose enter body
cells, lowering blood glucose levels to the normal
range. When blood sugar drops too low however, the
alpha cells secrete glucagon. This signals the
liver to release stored glycogen and change it
back to glucose, raising blood sugar levels to the
normal range. The result of the disturbed
metabolism of glucose causes an abnormal
accumulation of sugar in the blood stream and the
diabetic condition.
Characterization of Diabetes:
Diabetes is characterised by three well-known
syndromes, polydipsia (excessive thirst),
polyphagia (excessive hunger) and polyuria
(excessive urination). Laboratory findings
reveal high blood sugar and glucose in the urine
and as the metabolic derangement worsens,
excessive ketone bodies in the blood and urine.
The accumulation of these produces acidosis
which, if not counteracted, can result in coma
and death
There are three main types of
diabetes:
o Type 1 or 'Insulin-Dependent Diabetes
Mellitus' (IDDM) also known as 'Juvenile Onset
Diabetes'.
o Type 2 or 'Noninsulin-Dependent Diabetes
Mellitus' (NIDDM) also known as 'Adult Onset
Diabetes'.
o Gestational diabetes.
Type 1 Diabetes (Insulin-Dependent
Diabetes Mellitus/IDDM)
Insulin-dependent diabetes is considered an
autoimmune disease in which the immune system
attacks the insulinproducing beta cells in the
pancreas and destroys them. The pancreas
produces little or no insulin and it is then
almost certain that life-long insulin
replacement will be necessary. The exact
mechanism for the body's immune system attack to
the beta cells is unknown but the most likely
causes are viral infection, genetic factors and
free radicals.
Interest has been generated lately in the strong
evidence linking exposure to a protein in cow's
milk (bovine albumin peptide) in infancy to the
autoimmune response and subsequent Type 1
diabetes. In detailed studies1 it was shown that
patients with Type 1 diabetes were more likely
to have been breast-fed for less than three
months and to have been exposed to cow's milk or
solid foods before the age of four months. Since
the cow's milk protein can enter the mother's
breast milk, in cases of family history of
diabetes it is recommended that the mother avoid
cow's milk while breast-feeding.
IDDM accounts for about 5 to 10 percent of
diagnosed diabetes in the USA and develops most
often in children and young adults, but the
disorder can appear at any age. Symptoms usually
develop over a short period, although beta cell
destruction can begin months, even years,
earlier.
Over time both Type 1 and Type 2 diabetes are
accompanied by many severe complications, such
as blindness, renal failure, lower- limb
amputations, cardiovascular disease and stroke.
For those with Type 1 diabetes the object is not
to find a way to get off insulin but rather to
prevent the long-term complications. It is
encouraging to note that modern research has
demonstrated the amount of insulin required
could be reduced through appropriate life style
modifications and the likelihood of consequent
complications significantly lowered.
Type 2 Diabetes (Noninsulin-Dependent
Diabetes Mellitus/NIDDM)
The most common form of diabetes is
noninsulin-dependent diabetes. About 90 to 95
percent of people with diabetes have Type 2. In
the USA more than 16 million people, over 7% of
the adult population, have Type 2 with 600,000
new cases diagnosed each year. In many patients,
the initial diagnosis of Type 2 diabetes is
delayed perhaps by as much as 10 years because
symptoms are often absent or very mild during
its early stages.
Type 2 diabetes usually develops in adults over
the age of 40 and is most common among adults
over age 55. It is particularly common among the
elderly and in many minority populations,
including African Americans, Hispanic Americans,
American Indians and Asian and Pacific Islander
Americans, in whom it may occur in 10-50% of
adults.
Type 2 diabetics typically have elevated levels
of insulin, often producing two to three times
the normal amount. Rather than an insulin
deficiency condition it is an "insulin
resistance" condition whereby the body loses its
ability to properly respond to the signals given
by insulin. We now know that excess insulin
brought on by insulin resistance is not only
associated with elevated blood sugar levels, but
also with high blood pressure and increased
rates of atherosclerosis.
In the treatment of Type 2 diabetes, dietary
modification has been found to be of primary
importance and should be diligently followed
before using drug intervention since most cases
can be controlled by diet alone. For all Type 2
diabetics an effective treatment approach should
employ a broad-based therapeutic regimen. Such a
regimen would incorporate appropriate diet,
prescribed exercise, stress reduction techniques
and a substantial amount of specific nutritional
supplements. If adequate control of blood sugar
levels remains problematic, conventional
treatment with insulin and oral agents can be
initiated.
Gestational Diabetes
Gestational diabetes develops or is
discovered during pregnancy. This type usually
disappears when the pregnancy is over, but women
who have had gestational diabetes have a greater
risk of developing NIDDM later in their lives.
Presenting Symptoms
The clinical manifestations of diabetes in the order in which they usually appear are:
o frequent, copious urination
o excessive thirst
o rapid weight loss
o excessive hunger
o drowsiness, fatigue
o itching of the genitals and skin
o visual disturbances
o skin infections
o slow healing
o paraesthesia in the hands or feet
Other signs of diabetes include lingering influenza-like symptoms, loss of hair on the legs, increased facial hair, small yellow bumps anywhere on the body (known as xanthomas-cholesterol) and inflammation of the penile skin.
In most juvenile cases the earliest symptoms noted are increased urination, thirst and hunger. Other symptoms include irritability, nausea or vomiting, weakness and fatigue. Physical findings in the adult are mostly attributable to complications, and the first sign of the disease may be some dermatological, circulatory, neurological or visual complications.
